Non-Excoriated Eczematous Diseases - Skin Disorders

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Impetigo generally presents as a shallow, red erosion covered with crust. The presence of weeping, crusting, and pruritus makes it possible for the non bullous form of the illness to be morphologically placed within the group of eczematous diseases. Impetigo is identified by its location about the nose and mouth, by its rapid appearance and spread, by the presence of pus underneath the crust, and by its prompt response to antibacterial therapy. Other clues to correct diagnosis contain history of contagion, paucity of inflammation compared with the magnitude of crusting, and recovery of streptococcal or staphylococcal bacteria Oil culture.

The term infectious eczematoid dermatitis was originally employed for those eczematous lesions that appeared ill the skin about infected, draining lesions. Still, it is now apparent that this eczematization is due extra to the macerating and irritating impact of the draining fluid than to any bacteria that it may possibly include. Therefore the rash about an ileostomy (which drains sterile fluid) is related in look to that about a colostomy (which drains fluid with a high bacterial count). For this reason, this method can be viewed as a variant of weak-irritant get in touch with dermatitis the term "infectious eczematoid dermatitis" is top left as an historical footnote in the annals of dermatology.

The patches and plaques of perioral dermatitis are normally covered with a tiny amount of yellow scale. As in seborrheic dermatitis, a illness to which it is related, the yellow is due to the exudation of little amounts of serum onto the surface of the skin. Minute pustules might possibly stud the surface of the erythematous patches or plaques, and thus the condition is also regarded as . As the term "perioral" implies, this condition occurs on the lower half of the face. A characteristic function is the presence of a narrow margin of regular skin that occurs in between the lips and the beginning of the eruption.

There are many types of sunlight-induced eczematous diseases. Very first, photosensitivity may be induced by internally administered medications such as the tetracyclines, phenothiazines, thiazide diuretics, sulfonamide antibiotics, and nalidixic acid. Second, photocontact dermatitis could possibly occur in a tiny percentage of patients applying cosmetics containing musk ambrette, sunscreens containing para-raminobenzoic acid (PABA), PABA esters, cinnamates, or benzophenones and in the past at least, soaps containing halogenated bacteriostatic agents. Third, chronic sun exposure occurring more than years may possibly result in the development of hundreds of small, closely set, slightly crusted actinic keratoses superimposed on an inflammatory background. This method can be deemed conceptually as a form of actinic dermatitis. Lastly, the photosensitivity eruption of systemic lupus erythematosus (the "butterfly eruption") is oftentimes so intense it takes on an eczematous morphology in a sun-exposed distribution pattern.