Skin Disorders - Clinical Presentation

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Tinea capitis, or "ringworm" of the scalp, presents itself as 1 or extra sharply marginated plaques of partial alopecia. Inflammation and scale are present, but usually these two modifications are very minimal. The recognition of broken hairs (stubble and black dots at the follicular orifices) is the preferred clue to right diagnosis.

Almost all circumstances happen in kids, but the diagnosis really should be considered in any adult presenting with evidence of localized alopecia . Kerion formation is a complication that occurs in about 10% of situations. This represents a sensitization phenomenon whereby the fungi induce a remarkably brisk inflammatory reaction with resulting pustulation, crusting, and edema formation. Wood's lamp examination does not reveal fluorescence in the most prevalent forms of tinea capitis or in kerion formation. Sadly, KOH preparations are tough for the inexperienced to interpret. For this reason, any suspected diagnosis needs the plucking of infected hairs for fungal culture.

Course and Prognosis

Tinea capitis and zoophilic tinea corporis commonly resolve spontaneously immediately after 6 to 12 months of activity. Tinea pedis, tinea cruris, and anthropophilic tinea corporis continue indefinitely. There are, on the other hand, periods of relative quiescence and exacerbation. All of these fungal diseases respond properly to treatment, but with the exception of tinea capitis and zoophilic tinea corporis infections, recurrence following treatment is rather likely.

Pathogenesis

Tinea pedis, tinea cruris, and anthropophilic tinea corporis are most commonly brought on by Trichophyton rulnum. Trichophyton interdigitate and Epidermophhyton floccoswn infections are also noticed. Generally, one cannot predict the causative organism on the basis of clinical appearance. Zoophilic tinea corporis can be brought on by Microsporum canis, Trichophyton mentagrophytes, and Trichophyton verrucosum. Tinea capitis is caused by Trichophyton tonsurans in 90% of cases.

The likelihood of inoculation with any of these fungi is enhanced if cuts and scratches are present on the skin. Clinical evidence of infection following inoculation is enhanced by the presence of warmth and moisture, such as occurs in the groin and under footwear. Depression of cell-mediated immune responsiveness, as in atopic patients, is a key predisposing factor for the development of T. rubrum infection.

Therapy

Tinea cruris and those instances of tinea pedis that involve only the internet spaces can be treated with any of the topical antifungal agents. Other forms of tinea pedis often demand the use of griseofulvin. Mild circumstances of tinea corporis also respond well to topical agents. Extensive disease and those instances with a component of follicular involvement are preferred treated with griseofulvin. Tinea capitis calls for the use of griseofulvin. Orally administered ketoconazole therapy is rarely suitable for either tinea corporis or capitis. Kerion on nation, if present, can be treated with intralesional steroid injections or with a short burst of systemically administered steroids.